RT Book, Section A1 Fried, Isabella A1 Barnhill, Raymond L. A2 Barnhill, Raymond L. A2 Crowson, A. Neil A2 Magro, Cynthia M. A2 Piepkorn, Michael W. A2 Kutzner, Heinz A2 Desman, Garrett T. SR Print(0) ID 1178387863 T1 Psoriasiform Dermatitis T2 Barnhill's Dermatopathology, 4e YR 2020 FD 2020 PB McGraw Hill Education PP New York, NY SN 9780071828222 LK dermatology.mhmedical.com/content.aspx?aid=1178387863 RD 2024/03/28 AB Inflammatory dermatoses may be categorized based on the pattern of inflammation1 and epidermal changes.2 With psoriasis as the prototype, the psoriasiform dermatitides are characterized by regular elongation of rete ridges.3 This may result from increased keratinocyte proliferation due to more rapid turnover of stem cells or an increased pool of proliferating cells. Epidermal thickening may affect any or all of the layers of the epidermis. The stratum corneum may be thickened (hyperkeratosis) and may retain nuclear remnants (parakeratosis). Thickening of the granular layer or spinous layer is termed “hypergranulosis” or “acanthosis,” respectively.